By Joao H. Bastos
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Additional resources for Athlete Performance and Injuries
Additionally, type I muscle fibers, predominantly recruited in endurance sports and recognized by the predominance of aerobic oxidative metabolism, present an inverse relationship with CK-MM activity. , 2007). , 1997a; Dias et al, 2007). , 2006). a, 1997a). , 2006; Rivera et al. , 1997a). , 2007). However, because the nature of athletic performance is multigenic and multifactorial, further investigation becomes necessary, mainly due to the fact that investigations have examined these polymorphisms together with VO2max assessments and/or running economy, without, however, assessing serum concentrations of total CK or CK-MM.
3. , 1997). , 1998). During early stages of embryogenesis, GDF8 expression is restricted to the myotome compartment of developing somites. , 1998). , 2004). , 2007). , 2011). , 1998). Mice with null mutations of the myostatin gene have increased muscle mass. , 2007). In humans, a loss-of-function mutation in the myostatin gene in a child increased muscle bulk and strength. , 2004). , 2011). , 2004). , 2010). Also other genes within the myostatin pathway as well as regulatory elements in myostatin expression should be studied as candidate genes.
2007). , 2007), and genetic mapping with newer approaches such as genome-wide association may yield novel insights into the physiological responses to exercise. Over the last two decades, a number of groups have begun to investigate the influence of candidate gene polymorphisms on endurance performance and, arising from their studies, a large number of genetic variants have been well documented, showing association with physical performance-related phenotypes (Ostrander et al. 2009; Schoenfelder, 2010).